Search results for " interleukin 6"

showing 9 items of 9 documents

Mechanical stretch on endothelial cells interconnects innate and adaptive immune response in hypertension

2018

0301 basic medicinebiologyPhysiologybusiness.industryCellular differentiation030204 cardiovascular system & hematologySTAT3 Transcription FactorAcquired immune systemCell biologySTAT3 Intermediate monocyte Endothelium Interleukin 6 Nitric oxide03 medical and health scienceschemistry.chemical_compound030104 developmental biology0302 clinical medicinechemistryPhysiology (medical)biology.proteinMedicineCardiology and Cardiovascular MedicineHydrogen peroxidebusinessInterleukin 6
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Inflammation, genes and zinc in ageing and age-related diseases.

2006

Lifelong antigenic burden determines a condition of chronic inflammation, with increased lymphocyte activation and pro-inflammatory cytokine production. A large number of studies have documented changes in Zn metabolism in experimental animal models of acute and chronic inflammation and in human chronic inflammatory diseases. In particular, modification of zinc plasma concentration as well as intracellular disturbance of antioxidant intracellular pathways have been found associated to age-related inflammatory diseases, like atherosclerosis. Zinc deficiency is extremely diffused in aged people that are educated to avoid meat and other high Zn-content foods due to fear of cholesterol. Rather,…

Agingmedicine.medical_treatmentLongevityGene ExpressionInflammationBiologychemistry.chemical_compoundmedicinecytokine interleukin 6 metallothionein tumor necrosis factor alpha zincAnimalsHumansGeneTranscription factorCellular SenescenceInflammationPolymorphism GeneticCholesterolInterleukin-6Tumor Necrosis Factor-alphamedicine.diseaseAtherosclerosisImmunity InnateZincCytokinechemistryAgeingImmunologyZinc deficiencyCytokinesMetallothioneinGeriatrics and Gerontologymedicine.symptomGerontologyIntracellular
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AN IL-6/IL-6 SOLUBLE RECEPTOR (IL-6R) HYBRID PROTEIN (H-IL-6) INDUCES EPO-INDEPENDENT ERYTHROID DIFFERENTIATION IN HUMAN CD34+CELLS

2000

H-IL-6 is a hybrid protein constructed to contain IL-6 and its soluble receptor linked by a flexible peptide chain. Here we show that H-IL-6 strongly enhances proliferation of human CD34(+)cells in serum-free liquid culture, and that the majority of the cells generated belong to the erythroid lineage, being positive for the marker Glycophorin A. Conversely, H-IL-6 does not increase the number of myeloid, CD13-positive cells. Comparable effects are observed on progenitors from cord blood and adult peripheral blood. Therefore, H-IL-6 triggers an erythroid-inducing signal in haematopoietic progenitor cells, independently from erythropoietin (EPO).

ErythrocytesTime FactorsMyeloidCellular differentiationInterleukin 6Antigens CD34BiochemistryCulture Media Serum-FreeSerum-Freehemic and lymphatic diseasesReceptorsLeukocytesImmunology and AllergyErythropoiesisGlycophorinsStem Cell FactorbiologyChemistryCord bloodCell DifferentiationHematologyFetal BloodFlow CytometryEndothelial stem cellHaematopoiesismedicine.anatomical_structureGlycophorinCD34+medicine.drugRecombinant Fusion ProteinsMononuclearImmunologyCD13 AntigensmedicineHumansGlycophorinAntigensProgenitor cellErythropoietinMolecular BiologyInterleukin 3Interleukin-6CD34+; Cord blood; Erythropoiesis; Interleukin 6; Stem cell factor; Antigens CD34; CD13 Antigens; Cell Differentiation; Culture Media Serum-Free; Erythrocytes; Erythropoietin; Fetal Blood; Flow Cytometry; Glycophorin; Hematopoietic Stem Cells; Humans; Interleukin-6; Leukocytes Mononuclear; Peptides; Receptors Interleukin-6; Recombinant Fusion Proteins; Stem Cell Factor; Time Factors; Immunology and Allergy; Immunology; Biochemistry; Hematology; Molecular BiologyHematopoietic Stem CellsReceptors Interleukin-6Molecular biologyCulture MediaErythropoietinLeukocytes Mononuclearbiology.proteinCD34PeptidesCytokine
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Xanthine oxidase-induced oxidative stress causes activation of NF-κB and inflammation in the liver of type I diabetic rats

2009

We previously showed that xanthine oxidase activity increases in type I diabetic animals and that this is a significant cause of the oxidative stress which occurs in the disease. The aim of this work was to search for molecular links between xanthine oxidase-induced oxidative stress and inflammation in Type I diabetes and to assess the ability of allopurinol, a drug widely used in clinical practice, to prevent both processes. 3-month-old male Wistar rats were made diabetic by injection (i.p.) of either streptozotocin or alloxan. Allopurinol (32 mg/Kg) was administered (i.p) to diabetic rats after they had shown clear signs of diabetes such as glucosuria and polyuria. Hepatic phospho-IKKbeta…

MaleTranscriptional ActivationXanthine Oxidasemedicine.medical_specialtyNeutrophilsAllopurinolAllopurinolInterleukin 6Free radicalsInflammationmedicine.disease_causeBiochemistryStreptozocinDiabetes Mellitus ExperimentalDiabetic complicationsProinflammatory cytokineInterleukin 1βchemistry.chemical_compoundCell MovementPhysiology (medical)Internal medicineDiabetes mellitusAlloxanmedicineAnimalsRats WistarXanthine oxidasePolyuriabusiness.industryAllopurinol; Interleukin 1β; Interleukin 6; Diabetic complications; Free radicalsNF-kappa BXanthineStreptozotocinmedicine.diseaseRatsOxidative StressEndocrinologyLiverchemistryCytokinesInflammation Mediatorsmedicine.symptombusinessOxidative stressmedicine.drugFree Radical Biology and Medicine
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Interleukin-6 gene polymorphism is an age-dependent risk factor for myocardial infarction in men.

2005

Summary Several studies show that inflammatory components may contribute to atherosclerosis and increase the risk for myocardial infarction (MI). Interleukin-6 (IL-6) is a key pro-inflammatory and immune-modulatory cytokine of relevance for cardiovascular diseases. In this case-control study, 200 patients with MI and 257 healthy controls were genotyped for the polymorphism present in −174 promoter region of the IL-6 gene. Plasma concentrations of IL-6 and C-reactive protein (CRP) in a group of patients and controls were measured. The −174 C allele was associated with an increased risk of developing MI (OR = 2.886, c.i. = 1.801–4.624, P = 0.0001) in older patients, while no association was f…

Malemedicine.medical_specialtyGenotypemedicine.medical_treatmentImmunologyMyocardial InfarctionGastroenterologyPolymorphism Single NucleotidePolymorphism (computer science)Risk FactorsInternal medicineGenotypeGeneticsmedicineHumansGenetic Predisposition to DiseaseMyocardial infarctionRisk factorAllelePromoter Regions GeneticMolecular BiologyGeneGenetics (clinical)AllelesAgedbusiness.industryInterleukin-6Age FactorsPromoterGeneral MedicineMiddle Agedmedicine.diseaseC reactive protein DNA interleukin 6CytokineC-Reactive ProteinCase-Control StudiesImmunologybusiness
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Interleukin 6 plasma levels predict with high sensitivity and specificity coronary stenosis detected by coronary angiography

2007

SummaryIn recent years new biomarkers able to measure the coronary atherosclerotic burden have been investigated. The aim of the present study was: i) to measure plasma levels of four biomarkers: C reactive protein (CRP), soluble intercellular adhesion molecule-1 (sICAM-1), interleukin 6 (IL-6), 8-isosprostane (8-ISO),in a series of patients undergoing coronary angiography; ii) to assess the power of the biomarkers to predict critical coronary stenosis detected by angiography. The study population consisted of a group of 438 subjects undergoing coronary angiography; 160 patients with 0, 1, 2, or 3 critical vessels were selected, and biomarkers plasma levels were measured in plasma samples o…

Malemedicine.medical_specialtyPathologyCoronary AngiographyDinoprostSensitivity and SpecificitySeverity of Illness IndexCoronary artery diseasePredictive Value of TestsInternal medicineBlood plasmamedicineHumansInterleukin 6Agedmedicine.diagnostic_testbiologyInterleukin-6business.industryC-reactive proteinCoronary StenosisAcute-phase proteinCoronary artery disease coronary angiography biomarkers interleukin 6 C reactive protein 8-isoprostane sICAM-1HematologyMiddle AgedIntercellular Adhesion Molecule-1medicine.diseaseStenosisC-Reactive ProteinROC CurveAngiographybiology.proteinCardiologyPopulation studyFemalebusinessBiomarkers
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Generation and function of the soluble interleukin-6 receptor

1999

NeuronsBinding SitesInterleukin-6ChemistryRecombinant Fusion ProteinsAlternative splicingOsteoclastsReceptors Interleukin-6BiochemistryAlternative SplicingSoluble Interleukin 6 ReceptorSolubilityBiochemistryAnimalsHumansAmino Acid SequenceEndotheliumSolubilitySignal transductionReceptorPeptide sequenceFunction (biology)Signal TransductionBiochemical Society Transactions
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Smad7 controls resistance of colitogenic T cells to regulatory T cell-mediated suppression.

2008

Background & Aims Foxp3-expressing regulatory T cells (Tregs) play a key role in the maintenance of the gut immune homeostasis, and an intact transforming growth factor (TGF)-β signaling is required for their function. In inflammatory bowel disease (IBD), the TGF-β signaling is impaired because of high expression of the inhibitory molecule Smad7. Although no intrinsic defects in Tregs function have been shown in IBD, it is still unknown whether colitogenic T cells are susceptible to Treg-mediated suppression. In this study, we have investigated whether IBD mucosal CD4+ T cells are resistant to Tregs and whether Smad7 is involved in this process. Methods IBD lamina propria mononuclear cells …

antisense oligonucleotideCD4-Positive T-LymphocytesAdoptive cell transferT-Lymphocytesanimal cellCell CommunicationInbred C57BLT-Lymphocytes RegulatoryTransgenicMiceregulatory T lymphocyteCrohn DiseaseTransforming Growth Factor betamononuclear cellRAG1 proteinIntestinal MucosaenteritisCells CulturedMice KnockoutSettore MED/12 - GastroenterologiaCulturedintegumentary systemmedicine.diagnostic_testarticleGastroenterologyInterleukinhemic and immune systemsT helper cellColitisRegulatoryUp-Regulationmedicine.anatomical_structurepriority journalgamma interferonSignal TransductionRegulatory T cellColonCellsKnockoutanimal experimentinterleukin 6chemical and pharmacologic phenomenaMice TransgenicBiologyinterleukin 2Recombination-activating geneFlow cytometryProinflammatory cytokineSmad7 ProteinmedicineAnimalsHumanscontrolled studyhumanlamina propriamouseCell ProliferationHomeodomain ProteinsCD4+ T lymphocytenonhumanHepatologyAnimalflow cytometryhuman cellanimal cell culturetransgenic mouseMice Inbred C57BLDisease Models Animalantisense oligonucleotide; gamma interferon; interleukin 17; interleukin 2; interleukin 6; RAG1 protein; Smad7 protein; animal cell; animal cell culture; animal experiment; article; CD4+ T lymphocyte; cell proliferation; colitis; controlled study; enteritis; flow cytometry; human; human cell; knockout mouse; lamina propria; mononuclear cell; mouse; nonhuman; priority journal; regulatory T lymphocyte; transgenic mouse; Animals; CD4-Positive T-Lymphocytes; Cell Communication; Cell Proliferation; Cells Cultured; Colitis; Colon; Crohn Disease; Disease Models Animal; Homeodomain Proteins; Humans; Intestinal Mucosa; Mice; Mice Inbred C57BL; Mice Knockout; Mice Transgenic; Signal Transduction; Smad7 Protein; T-Lymphocytes Regulatory; Transforming Growth Factor beta; Up-RegulationDisease ModelsImmunologyinterleukin 17knockout mouseTransforming growth factorGastroenterology
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Gene Therapy of Human Melanoma. Immunization of Patients with Autologous Tumor Cells Admixed with Allogeneic Melanoma Cells Secreting Interleukin 6 a…

1995

biologybusiness.industryMelanomaGenetic enhancementCancermedicine.diseaseSoluble Interleukin 6 ReceptorImmunizationImmunologyGeneticsbiology.proteinmedicineMolecular MedicineHuman melanomaInterleukin 6businessMolecular BiologyAutologous tumorHuman Gene Therapy
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